Ralf Köhler
Genetic deficit of SK3 and IK1 channels disrupts the endothelium-derived hyperpolarizing factor vasodilator pathway and causes hypertension.
Brähler S, Kaistha A, Schmidt V, Wölfle S, Busch C, Kaistha B, Kacik M, Hasenau A, Grgic I, Siiskonen H, Bond C, Adelman J, Wulff H, de Wit C, Hoyer J, Köhler R. Genetic deficit of SK3 and IK1 channels disrupts the endothelium-derived hyperpolarizing factor vasodilator pathway and causes hypertension. Circulation 2009; 119:2323-32.
20.04.2009Genetic deficit of SK3 and IK1 channels disrupts the endothelium-derived hyperpolarizing factor vasodilator pathway and causes hypertension.
20.04.2009Circulation 2009; 119:2323-32
Brähler Sebastian, Kaistha Anuradha, Schmidt Volker, Wölfle Stephanie E, Busch Christoph, Kaistha Brajesh P, Kacik Michael, Hasenau Anna-Lena, Grgic Ivica, Siiskonen Hanna, Bond Chris T, Adelman John P, Wulff Heike, de Wit Cor, Hoyer Joachim, Köhler Ralf
Prominent role of KCa3.1 in endothelium-derived hyperpolarizing factor-type dilations and conducted responses in the microcirculation in vivo.
Wölfle S, Schmidt V, Hoyer J, Köhler R, de Wit C. Prominent role of KCa3.1 in endothelium-derived hyperpolarizing factor-type dilations and conducted responses in the microcirculation in vivo. Cardiovasc Res 2009; 82:476-83.
13.02.2009Prominent role of KCa3.1 in endothelium-derived hyperpolarizing factor-type dilations and conducted responses in the microcirculation in vivo.
13.02.2009Cardiovasc Res 2009; 82:476-83
Wölfle Stephanie E, Schmidt Volker, Hoyer Joachim, Köhler Ralf, de Wit Cor