Cornelia Tillack
Development of a uniform, very aggressive disease phenotype in all homozygous carriers of the NOD2 mutation p.Leu1007fsX1008 with Crohn's disease and active smoking status resulting in ileal stenosis requiring surgery.
Schnitzler F, Friedrich M, Angelberger M, Diegelmann J, Stallhofer J, Wolf C, Dütschler J, Truniger S, Olszak T, Beigel F, Tillack C, Lohse P, Brand S. Development of a uniform, very aggressive disease phenotype in all homozygous carriers of the NOD2 mutation p.Leu1007fsX1008 with Crohn's disease and active smoking status resulting in ileal stenosis requiring surgery. PloS one 2020; 15:e0236421.
27.07.2020Development of a uniform, very aggressive disease phenotype in all homozygous carriers of the NOD2 mutation p.Leu1007fsX1008 with Crohn's disease and active smoking status resulting in ileal stenosis requiring surgery.
27.07.2020PloS one 2020; 15:e0236421
Schnitzler Fabian, Friedrich Matthias, Angelberger Marianne, Diegelmann Julia, Stallhofer Johannes, Wolf Christiane, Dütschler Joel, Truniger Samuel, Olszak Torsten, Beigel Florian, Tillack Cornelia, Lohse Peter, Brand Stephan
Solid Organ Transplantation in Patients with Inflammatory Bowel Diseases (IBD): Analysis of Transplantation Outcome and IBD Activity in a Large Single Center Cohort
Schnitzler F, Grüner N, Rust C, Guba M, Denk G, Zachoval R, Göke B, Tillack C, Beigel F, Olszak T, Angelberger M, Wolf C, Karbalai N, Habicht A, Fischereder M, Schönermarck U, Stallhofer J, Friedrich M, Brand S. Solid Organ Transplantation in Patients with Inflammatory Bowel Diseases (IBD): Analysis of Transplantation Outcome and IBD Activity in a Large Single Center Cohort. PloS one 2015; 10:e0135807.
19.08.2015Solid Organ Transplantation in Patients with Inflammatory Bowel Diseases (IBD): Analysis of Transplantation Outcome and IBD Activity in a Large Single Center Cohort
19.08.2015PloS one 2015; 10:e0135807
Schnitzler Fabian, Grüner Norbert, Rust Christian, Guba Markus, Denk Gerald, Zachoval Reinhart, Göke Burkhard, Tillack Cornelia, Beigel Florian, Olszak Torsten, Angelberger Marianne, Wolf Christiane, Karbalai Nazanin, Habicht Antje, Fischereder Michael, Schönermarck Ulf, Stallhofer Johannes, Friedrich Matthias, Brand Stephan
The NOD2 Single Nucleotide Polymorphism rs72796353 (IVS4+10 A>C) Is a Predictor for Perianal Fistulas in Patients with Crohn's Disease in the Absence of Other NOD2 Mutations
Schnitzler F, Lohse P, Glas J, Göke B, Beigel F, Tillack C, Olszak T, Diegelmann J, Angelberger M, Stallhofer J, Wolf C, Friedrich M, Brand S. The NOD2 Single Nucleotide Polymorphism rs72796353 (IVS4+10 A>C) Is a Predictor for Perianal Fistulas in Patients with Crohn's Disease in the Absence of Other NOD2 Mutations. PloS one 2015; 10:e0116044.
06.07.2015The NOD2 Single Nucleotide Polymorphism rs72796353 (IVS4+10 A>C) Is a Predictor for Perianal Fistulas in Patients with Crohn's Disease in the Absence of Other NOD2 Mutations
06.07.2015PloS one 2015; 10:e0116044
Schnitzler Fabian, Lohse Peter, Glas Jürgen, Göke Burkhard, Beigel Florian, Tillack Cornelia, Olszak Torsten, Diegelmann Julia, Angelberger Marianne, Stallhofer Johannes, Wolf Christiane, Friedrich Matthias, Brand Stephan
The NOD2 p.Leu1007fsX1008 mutation (rs2066847) is a stronger predictor of the clinical course of Crohn's disease than the FOXO3A intron variant rs12212067
Schnitzler F, Lohse P, Glas J, Göke B, Stallhofer J, Tillack C, Beigel F, Olszak T, Diegelmann J, Angelberger M, Wolf C, Friedrich M, Brand S. The NOD2 p.Leu1007fsX1008 mutation (rs2066847) is a stronger predictor of the clinical course of Crohn's disease than the FOXO3A intron variant rs12212067. PloS one 2014; 9:e108503.
03.11.2014The NOD2 p.Leu1007fsX1008 mutation (rs2066847) is a stronger predictor of the clinical course of Crohn's disease than the FOXO3A intron variant rs12212067
03.11.2014PloS one 2014; 9:e108503
Schnitzler Fabian, Lohse Peter, Glas Jürgen, Göke Burkhard, Stallhofer Johannes, Tillack Cornelia, Beigel Florian, Olszak Torsten, Diegelmann Julia, Angelberger Marianne, Wolf Christiane, Friedrich Matthias, Brand Stephan
IL-36γ sustains a proinflammatory self-amplifying loop with IL-17C in anti-TNF-induced psoriasiform skin lesions of patients with Crohn's disease
Friedrich M, Tillack C, Wollenberg A, Schauber J, Brand S. IL-36γ sustains a proinflammatory self-amplifying loop with IL-17C in anti-TNF-induced psoriasiform skin lesions of patients with Crohn's disease. Inflamm Bowel Dis 2014; 20:1891-901.
01.11.2014IL-36γ sustains a proinflammatory self-amplifying loop with IL-17C in anti-TNF-induced psoriasiform skin lesions of patients with Crohn's disease
01.11.2014Inflamm Bowel Dis 2014; 20:1891-901
Friedrich Matthias, Tillack Cornelia, Wollenberg Andreas, Schauber Jürgen, Brand Stephan
Risk of malignancies in patients with inflammatory bowel disease treated with thiopurines or anti-TNF alpha antibodies
Beigel F, Brand S, Seiderer J, Göke B, Laubender R, Van Steen K, John J, Breiteneicher S, Tillack C, Schnitzler F, Steinborn A, Ochsenkühn T. Risk of malignancies in patients with inflammatory bowel disease treated with thiopurines or anti-TNF alpha antibodies. Pharmacoepidemiol Drug Saf 2014; 23:735-44.
30.04.2014Risk of malignancies in patients with inflammatory bowel disease treated with thiopurines or anti-TNF alpha antibodies
30.04.2014Pharmacoepidemiol Drug Saf 2014; 23:735-44
Beigel Florian, Brand Stephan, Seiderer Julia, Göke Burkhard, Laubender Rüdiger P, Van Steen Kristel, John Jestinah Mahachie, Breiteneicher Simone, Tillack Cornelia, Schnitzler Fabian, Steinborn Anni, Ochsenkühn Thomas
Anti-TNF antibody-induced psoriasiform skin lesions in patients with inflammatory bowel disease are characterised by interferon-γ-expressing Th1 cells and IL-17A/IL-22-expressing Th17 cells and respond to anti-IL-12/IL-23 antibody treatment
Tillack C, Koburger M, Wagner J, Glas J, Diegelmann J, Koglin S, Dombrowski Y, Schauber J, Wollenberg A, Maier H, Wetzke M, Ehmann L, Friedrich M, Laubender R, Papay P, Vogelsang H, Stallhofer J, Beigel F, Bedynek A, Brand S. Anti-TNF antibody-induced psoriasiform skin lesions in patients with inflammatory bowel disease are characterised by interferon-γ-expressing Th1 cells and IL-17A/IL-22-expressing Th17 cells and respond to anti-IL-12/IL-23 antibody treatment. Gut 2013; 63:567-77.
06.03.2013Anti-TNF antibody-induced psoriasiform skin lesions in patients with inflammatory bowel disease are characterised by interferon-γ-expressing Th1 cells and IL-17A/IL-22-expressing Th17 cells and respond to anti-IL-12/IL-23 antibody treatment
06.03.2013Gut 2013; 63:567-77
Tillack Cornelia, Koburger Maria, Wagner Johanna, Glas Jürgen, Diegelmann Julia, Koglin Sarah, Dombrowski Yvonne, Schauber Jürgen, Wollenberg Andreas, Maier Harald, Wetzke Martin, Ehmann Laura Maximiliane, Friedrich Matthias, Laubender Rüdiger P, Papay Pavol, Vogelsang Harald, Stallhofer Johannes, Beigel Florian, Bedynek Andrea, Brand Stephan
Analysis of IL12B gene variants in inflammatory bowel disease
Glas J, Czamara D, Diegelmann J, Ochsenkühn T, Göke B, Wetzke M, Steib C, Stallhofer J, Beigel F, Friedrich M, Tillack C, Fries C, Olszak T, Wagner J, Seiderer J, Brand S. Analysis of IL12B gene variants in inflammatory bowel disease. PloS one 2012; 7:e34349.
30.03.2012Analysis of IL12B gene variants in inflammatory bowel disease
30.03.2012PloS one 2012; 7:e34349
Glas Jürgen, Czamara Darina, Diegelmann Julia, Ochsenkühn Thomas, Göke Burkhard, Wetzke Martin, Steib Christian, Stallhofer Johannes, Beigel Florian, Friedrich Matthias, Tillack Cornelia, Fries Christoph, Olszak Torsten, Wagner Johanna, Seiderer Julia, Brand Stephan
PTPN2 gene variants are associated with susceptibility to both Crohn's disease and ulcerative colitis supporting a common genetic disease background
Glas J, Czamara D, Diegelmann J, Karbalai N, Ochsenkühn T, Göke B, Steib C, Friedrich M, Stallhofer J, Tillack C, Beigel F, Wetzke M, Olszak T, Seiderer J, Wagner J, Brand S. PTPN2 gene variants are associated with susceptibility to both Crohn's disease and ulcerative colitis supporting a common genetic disease background. PloS one 2012; 7:e33682.
21.03.2012PTPN2 gene variants are associated with susceptibility to both Crohn's disease and ulcerative colitis supporting a common genetic disease background
21.03.2012PloS one 2012; 7:e33682
Glas Jürgen, Czamara Darina, Diegelmann Julia, Karbalai Nazanin, Ochsenkühn Thomas, Göke Burkhard, Steib Christian, Friedrich Matthias, Stallhofer Johannes, Tillack Cornelia, Beigel Florian, Wetzke Martin, Olszak Torsten, Seiderer Julia, Wagner Johanna, Brand Stephan
The role of osteopontin (OPN/SPP1) haplotypes in the susceptibility to Crohn's disease
Glas J, Czamara D, Diegelmann J, Friedrich M, Steib C, Beigel F, Olszak T, Tillack C, Fries C, Wetzke M, Bayrle C, Seiderer J, Brand S. The role of osteopontin (OPN/SPP1) haplotypes in the susceptibility to Crohn's disease. PloS one 2011; 6:e29309.
29.12.2011The role of osteopontin (OPN/SPP1) haplotypes in the susceptibility to Crohn's disease
29.12.2011PloS one 2011; 6:e29309
Glas Jürgen, Czamara Darina, Diegelmann Julia, Friedrich Matthias, Steib Christian, Beigel Florian, Olszak Torsten, Tillack Cornelia, Fries Christoph, Wetzke Martin, Bayrle Corinna, Seiderer Julia, Brand Stephan
Iron status and analysis of efficacy and safety of ferric carboxymaltose treatment in patients with inflammatory bowel disease
Beigel F, Ochsenkühn T, Seiderer J, Göke B, Weidinger M, Breiteneicher S, Schnitzler F, Tillack C, Laubender R, Löhr B, Brand S. Iron status and analysis of efficacy and safety of ferric carboxymaltose treatment in patients with inflammatory bowel disease. Digestion 2011; 85:47-54.
14.12.2011Iron status and analysis of efficacy and safety of ferric carboxymaltose treatment in patients with inflammatory bowel disease
14.12.2011Digestion 2011; 85:47-54
Beigel Florian, Ochsenkühn Thomas, Seiderer Julia, Göke Burkhard, Weidinger Maria, Breiteneicher Simone, Schnitzler Fabian, Tillack Cornelia, Laubender Rüdiger P, Löhr Beate, Brand Stephan
CEACAM6 gene variants in inflammatory bowel disease
Glas J, Czamara D, Diegelmann J, Müller-Myhsok B, Lohse P, Wolf C, Ochsenkühn T, Göke B, Lass U, Olszak T, Beigel F, Weidinger M, Pfennig S, Tillack C, Fries C, Seiderer J, Brand S. CEACAM6 gene variants in inflammatory bowel disease. PloS one 2011; 6:e19319.
29.04.2011CEACAM6 gene variants in inflammatory bowel disease
29.04.2011PloS one 2011; 6:e19319
Glas Jürgen, Czamara Darina, Diegelmann Julia, Müller-Myhsok Bertram, Lohse Peter, Wolf Christiane, Ochsenkühn Thomas, Göke Burkhard, Lass Ulrich, Olszak Torsten, Beigel Florian, Weidinger Maria, Pfennig Simone, Tillack Cornelia, Fries Christoph, Seiderer Julia, Brand Stephan
The NOD2 single nucleotide polymorphisms rs2066843 and rs2076756 are novel and common Crohn's disease susceptibility gene variants
Glas J, Czamara D, Diegelmann J, Lohse P, Ochsenkühn T, Göke B, Müller-Myhsok B, Weidinger M, Laubender R, Olszak T, Jürgens M, Beigel F, Pfennig S, Tillack C, Seiderer J, Brand S. The NOD2 single nucleotide polymorphisms rs2066843 and rs2076756 are novel and common Crohn's disease susceptibility gene variants. PloS one 2010; 5:e14466.
30.12.2010The NOD2 single nucleotide polymorphisms rs2066843 and rs2076756 are novel and common Crohn's disease susceptibility gene variants
30.12.2010PloS one 2010; 5:e14466
Glas Jürgen, Czamara Darina, Diegelmann Julia, Lohse Peter, Ochsenkühn Thomas, Göke Burkhard, Müller-Myhsok Bertram, Weidinger Maria, Laubender Rüdiger P, Olszak Torsten, Jürgens Matthias, Beigel Florian, Pfennig Simone, Tillack Cornelia, Seiderer Julia, Brand Stephan
Functional SFTPD gene variants are not associated with susceptibility to inflammatory bowel disease in the German population
Glas J, Seiderer J, Tillack C, Paschos E, Wetzke M, Diegelmann J, Czamara D, Brand S. Functional SFTPD gene variants are not associated with susceptibility to inflammatory bowel disease in the German population. Inflamm Bowel Dis 2010; 17:1439-40.
03.12.2010Functional SFTPD gene variants are not associated with susceptibility to inflammatory bowel disease in the German population
03.12.2010Inflamm Bowel Dis 2010; 17:1439-40
Glas Jürgen, Seiderer Julia, Tillack Cornelia, Paschos Ekaterini, Wetzke Martin, Diegelmann Julia, Czamara Darina, Brand Stephan
Safety of adalimumab in Crohn's disease during pregnancy: case report and review of the literature
Jürgens M, Ochsenkühn T, Göke B, Tillack C, Beigel F, Hasbargen U, Hübener C, Filik L, Brand S, Seiderer J. Safety of adalimumab in Crohn's disease during pregnancy: case report and review of the literature. Inflamm Bowel Dis 2010; 16:1634-6.
01.10.2010Safety of adalimumab in Crohn's disease during pregnancy: case report and review of the literature
01.10.2010Inflamm Bowel Dis 2010; 16:1634-6
Jürgens Matthias, Ochsenkühn Thomas, Göke Burkhard, Tillack Cornelia, Beigel Florian, Hasbargen Uwe, Hübener Christoph, Filik Levent, Brand Stephan, Seiderer Julia
The presence of fistulas and NOD2 homozygosity strongly predict intestinal stenosis in Crohn's disease independent of the IL23R genotype
Jürgens M, Herrmann K, Lohse P, Göke B, Kreis M, Schnitzler F, Weidinger M, Beigel F, Tillack C, Pfennig S, Wagner J, Wetzke M, Glas J, Seiderer J, Laubender R, Brand S, Ochsenkühn T. The presence of fistulas and NOD2 homozygosity strongly predict intestinal stenosis in Crohn's disease independent of the IL23R genotype. J Gastroenterol 2010; 45:721-31.
29.04.2010The presence of fistulas and NOD2 homozygosity strongly predict intestinal stenosis in Crohn's disease independent of the IL23R genotype
29.04.2010J Gastroenterol 2010; 45:721-31
Jürgens Matthias, Herrmann Karin, Lohse Peter, Göke Burkhard, Kreis Martin E, Schnitzler Fabian, Weidinger Maria, Beigel Florian, Tillack Cornelia, Pfennig Simone, Wagner Johanna, Wetzke Martin, Glas Jürgen, Seiderer Julia, Laubender Rüdiger P, Brand Stephan, Ochsenkühn Thomas
Disease activity, ANCA, and IL23R genotype status determine early response to infliximab in patients with ulcerative colitis
Jürgens M, Ochsenkühn T, Glas J, Göke B, Lohse P, Tillack C, Schnitzler F, Stallhofer J, Pfennig S, Beigel F, Wetzke M, Wagner J, Seiderer J, Weidinger M, Hartl F, Laubender R, Brand S. Disease activity, ANCA, and IL23R genotype status determine early response to infliximab in patients with ulcerative colitis. Am J Gastroenterol 2010; 105:1811-9.
02.03.2010Disease activity, ANCA, and IL23R genotype status determine early response to infliximab in patients with ulcerative colitis
02.03.2010Am J Gastroenterol 2010; 105:1811-9
Jürgens Matthias, Ochsenkühn Thomas, Glas Jürgen, Göke Burkhard, Lohse Peter, Tillack Cornelia, Schnitzler Fabian, Stallhofer Johannes, Pfennig Simone, Beigel Florian, Wetzke Martin, Wagner Johanna, Seiderer Julia, Weidinger Maria, Hartl Franziska, Laubender Rüdiger P, Brand Stephan
Hepatosplenic T-cell lymphoma in a patient with Crohn's disease
Beigel F, Jürgens M, Tillack C, Subklewe M, Mayr D, Göke B, Brand S, Ochsenkühn T. Hepatosplenic T-cell lymphoma in a patient with Crohn's disease. Nat Rev Gastroenterol Hepatol 2009; 6:433-6.
01.07.2009Hepatosplenic T-cell lymphoma in a patient with Crohn's disease
01.07.2009Nat Rev Gastroenterol Hepatol 2009; 6:433-6
Beigel Florian, Jürgens Matthias, Tillack Cornelia, Subklewe Marion, Mayr Doris, Göke Burkhard, Brand Stephan, Ochsenkühn Thomas
rs224136 on chromosome 10q21.1 and variants in PHOX2B, NCF4, and FAM92B are not major genetic risk factors for susceptibility to Crohn's disease in the German population
Glas J, Klein W, Epplen J, Schiemann U, Mussack T, Lohse P, Göke B, Ochsenkühn T, Folwaczny M, Müller-Myhsok B, Griga T, Jürgens M, Stallhofer J, Seiderer J, Pasciuto G, Tillack C, Diegelmann J, Pfennig S, Konrad A, Schmechel S, Wetzke M, Török H, Brand S. rs224136 on chromosome 10q21.1 and variants in PHOX2B, NCF4, and FAM92B are not major genetic risk factors for susceptibility to Crohn's disease in the German population. Am J Gastroenterol 2009; 104:665-72.
03.02.2009rs224136 on chromosome 10q21.1 and variants in PHOX2B, NCF4, and FAM92B are not major genetic risk factors for susceptibility to Crohn's disease in the German population
03.02.2009Am J Gastroenterol 2009; 104:665-72
Glas Jürgen, Klein Wolfram, Epplen Jörg T, Schiemann Uwe, Mussack Thomas, Lohse Peter, Göke Burkhard, Ochsenkühn Thomas, Folwaczny Matthias, Müller-Myhsok Bertram, Griga Thomas, Jürgens Matthias, Stallhofer Johannes, Seiderer Julia, Pasciuto Giulia, Tillack Cornelia, Diegelmann Julia, Pfennig Simone, Konrad Astrid, Schmechel Silke, Wetzke Martin, Török Helga-Paula, Brand Stephan
Correlation of magnetic resonance enteroclysis (MRE) and wireless capsule endoscopy (CE) in the diagnosis of small bowel lesions in Crohn's disease
Tillack C, Seiderer J, Brand S, Göke B, Reiser M, Schaefer C, Diepolder H, Ochsenkühn T, Herrmann K. Correlation of magnetic resonance enteroclysis (MRE) and wireless capsule endoscopy (CE) in the diagnosis of small bowel lesions in Crohn's disease. Inflamm Bowel Dis 2008; 14:1219-28.
01.09.2008Correlation of magnetic resonance enteroclysis (MRE) and wireless capsule endoscopy (CE) in the diagnosis of small bowel lesions in Crohn's disease
01.09.2008Inflamm Bowel Dis 2008; 14:1219-28
Tillack Cornelia, Seiderer Julia, Brand Stephan, Göke Burkhard, Reiser Maximilian F, Schaefer Claus, Diepolder Helmut, Ochsenkühn Thomas, Herrmann Karin A