Cor de Wit
Expression of Connexin43 Stimulates Endothelial Angiogenesis Independently of Gap Junctional Communication In Vitro.
Koepple C, Zhou Z, Huber L, Schulte M, Schmidt K, Gloe T, Kneser U, Schmidt V, de Wit C. Expression of Connexin43 Stimulates Endothelial Angiogenesis Independently of Gap Junctional Communication In Vitro. Int J Mol Sci 2021; 22
09.07.2021Expression of Connexin43 Stimulates Endothelial Angiogenesis Independently of Gap Junctional Communication In Vitro.
09.07.2021Int J Mol Sci 2021; 22
Koepple Christoph, Zhou Zizi, Huber Lena, Schulte Matthias, Schmidt Kjestine, Gloe Torsten, Kneser Ulrich, Schmidt Volker, de Wit Cor
Flow increase is decisive to initiate angiogenesis in veins exposed to altered hemodynamics.
Schmidt V, Hilgert J, Covi J, Leibig N, Wietbrock J, Arkudas A, Polykandriotis E, de Wit C, Horch R, Kneser U. Flow increase is decisive to initiate angiogenesis in veins exposed to altered hemodynamics. PloS one 2015; 10:e0117407.
30.01.2015Flow increase is decisive to initiate angiogenesis in veins exposed to altered hemodynamics.
30.01.2015PloS one 2015; 10:e0117407
Schmidt Volker, Hilgert Johannes G, Covi Jennifer M, Leibig Nico, Wietbrock Johanna O, Arkudas Andreas, Polykandriotis Elias, de Wit Cor, Horch Raymund E, Kneser Ulrich
High flow conditions increase connexin43 expression in a rat arteriovenous and angioinductive loop model.
Schmidt V, Hilgert J, Covi J, Weiss C, Wietbrock J, de Wit C, Horch R, Kneser U. High flow conditions increase connexin43 expression in a rat arteriovenous and angioinductive loop model. PloS one 2013; 8:e78782.
13.11.2013High flow conditions increase connexin43 expression in a rat arteriovenous and angioinductive loop model.
13.11.2013PloS one 2013; 8:e78782
Schmidt Volker, Hilgert Johannes G, Covi Jennifer M, Weiss Christian, Wietbrock Johanna O, de Wit Cor, Horch Raymund E, Kneser Ulrich
Defective Cx40 maintains Cx37 expression but intact Cx40 is crucial for conducted dilations irrespective of hypertension.
Jobs A, Schmidt K, Schmidt V, Lübkemeier I, van Veen T, Kurtz A, Willecke K, de Wit C. Defective Cx40 maintains Cx37 expression but intact Cx40 is crucial for conducted dilations irrespective of hypertension. Hypertension 2012; 60:1422-9.
22.10.2012Defective Cx40 maintains Cx37 expression but intact Cx40 is crucial for conducted dilations irrespective of hypertension.
22.10.2012Hypertension 2012; 60:1422-9
Jobs Alexander, Schmidt Kjestine, Schmidt Volker, Lübkemeier Indra, van Veen Toon A B, Kurtz Armin, Willecke Klaus, de Wit Cor
Connexin45 is expressed in vascular smooth muscle but its function remains elusive.
Schmidt V, Jobs A, von Maltzahn J, Wörsdörfer P, Willecke K, de Wit C. Connexin45 is expressed in vascular smooth muscle but its function remains elusive. PloS one 2012; 7:e42287.
27.07.2012Connexin45 is expressed in vascular smooth muscle but its function remains elusive.
27.07.2012PloS one 2012; 7:e42287
Schmidt Volker, Jobs Alexander, von Maltzahn Julia, Wörsdörfer Philipp, Willecke Klaus, de Wit Cor
Genetic deficit of SK3 and IK1 channels disrupts the endothelium-derived hyperpolarizing factor vasodilator pathway and causes hypertension.
Brähler S, Kaistha A, Schmidt V, Wölfle S, Busch C, Kaistha B, Kacik M, Hasenau A, Grgic I, Siiskonen H, Bond C, Adelman J, Wulff H, de Wit C, Hoyer J, Köhler R. Genetic deficit of SK3 and IK1 channels disrupts the endothelium-derived hyperpolarizing factor vasodilator pathway and causes hypertension. Circulation 2009; 119:2323-32.
20.04.2009Genetic deficit of SK3 and IK1 channels disrupts the endothelium-derived hyperpolarizing factor vasodilator pathway and causes hypertension.
20.04.2009Circulation 2009; 119:2323-32
Brähler Sebastian, Kaistha Anuradha, Schmidt Volker, Wölfle Stephanie E, Busch Christoph, Kaistha Brajesh P, Kacik Michael, Hasenau Anna-Lena, Grgic Ivica, Siiskonen Hanna, Bond Chris T, Adelman John P, Wulff Heike, de Wit Cor, Hoyer Joachim, Köhler Ralf
Prominent role of KCa3.1 in endothelium-derived hyperpolarizing factor-type dilations and conducted responses in the microcirculation in vivo.
Wölfle S, Schmidt V, Hoyer J, Köhler R, de Wit C. Prominent role of KCa3.1 in endothelium-derived hyperpolarizing factor-type dilations and conducted responses in the microcirculation in vivo. Cardiovasc Res 2009; 82:476-83.
13.02.2009Prominent role of KCa3.1 in endothelium-derived hyperpolarizing factor-type dilations and conducted responses in the microcirculation in vivo.
13.02.2009Cardiovasc Res 2009; 82:476-83
Wölfle Stephanie E, Schmidt Volker, Hoyer Joachim, Köhler Ralf, de Wit Cor
Signaling across myoendothelial gap junctions--fact or fiction?
de Wit C, Boettcher M, Schmidt V. Signaling across myoendothelial gap junctions--fact or fiction?. Cell Commun Adhes 2008; 15:231-45.
01.09.2008Signaling across myoendothelial gap junctions--fact or fiction?
01.09.2008Cell Commun Adhes 2008; 15:231-45
de Wit Cor, Boettcher Markus, Schmidt Volker
Gap junctions synchronize vascular tone within the microcirculation.
Schmidt V, Wölfle S, Boettcher M, de Wit C. Gap junctions synchronize vascular tone within the microcirculation. Pharmacol Rep 2008; 60:68-74.
01.01.2008Gap junctions synchronize vascular tone within the microcirculation.
01.01.2008Pharmacol Rep 2008; 60:68-74
Schmidt Volker, Wölfle Stephanie E, Boettcher Markus, de Wit Cor
Connexin45 cannot replace the function of connexin40 in conducting endothelium-dependent dilations along arterioles.
Wölfle S, Schmidt V, Hoepfl B, Gebert A, Alcoléa S, Gros D, de Wit C. Connexin45 cannot replace the function of connexin40 in conducting endothelium-dependent dilations along arterioles. Circ Res 2007; 101:1292-9.
11.10.2007Connexin45 cannot replace the function of connexin40 in conducting endothelium-dependent dilations along arterioles.
11.10.2007Circ Res 2007; 101:1292-9
Wölfle Stephanie E, Schmidt Volker, Hoepfl Bernd, Gebert Andreas, Alcoléa Sébastien, Gros Daniel, de Wit Cor