Publication

Leber's hereditary optic neuropathy mitochondrial DNA mutations in familial multiple sclerosis

Journal Paper/Review - Apr 1, 1999

Units
PubMed

Citation
Mojon D, Fujihara K, Hirano M, Miller C, Lincoff N, Jacobs L, Greenberg S. Leber's hereditary optic neuropathy mitochondrial DNA mutations in familial multiple sclerosis. Graefe's archive for clinical and experimental ophthalmology = Albrecht von Graefes Archiv für klinische und experimentelle Ophthalmologie 1999; 237:348-50.
Type
Journal Paper/Review (English)
Journal
Graefe's archive for clinical and experimental ophthalmology = Albrecht von Graefes Archiv für klinische und experimentelle Ophthalmologie 1999; 237
Publication Date
Apr 1, 1999
Issn Print
0721-832X
Pages
348-50
Brief description/objective

Leber's hereditary optic neuropathy (LHON) can be difficult to distinguish from optic neuritis due to multiple sclerosis (MS). For several decades an association of LHON and MS has been suspected, and within the past 7 years the LHON nucleotide (nt)-3460 and nt-11778 mtDNA mutations have been identified in several patients with MS-like phenotypes. To further study this association, we tested 42 index patients with clinically definite, familial MS for the LHON mtDNA mutations at nt-3460, nt-11778, and nt-14484. No patients had a pathogenic LHON mtDNA mutation; however, two MS patients with unilateral optic neuritis harbored the nt-15257 mtDNA polymorphism that was reported originally as a pathogenic LHON mutation. Several investigators have shown evidence that the nt-15257 mtDNA mutation is not primarily pathogenic. Therefore, we conclude that pathogenic LHON mtDNA mutations are absent or rare in unselected patients with familial, clinically definite MS (95% confidence intervals for each of the negative mutations 0-7.0%).